In 1954, Dubin and Johnson described a new clinicopathological entity consisting of chronic idiopathic jaundice with unidentified pigment in the liver in 12 cases. In the same year, Sprinz and Nelson published a report of four cases with persistent non-hemolytic hyperbilirubinemia associated with lipochrome-like pigment in the liver cells; soon, the syndrome had come to be known as Dubin Johnson syndrome (DJS). This relatively rare disorder is a variety of hereditary hyperbilirubinemia and is characterized by low-grade elevation of conjugated bilirubin and no other signs of hepatic injury. This activity describes the pathophysiology of Dubin johnson syndrome and highlights the role of the interprofessional team in its management.
- Provider:StatPearls, LLC
- Activity Link: https://www.statpearls.com/ArticleLibrary/viewarticle/20746
- Start Date: 2023-09-01 05:00:00
- End Date: 2023-09-01 05:00:00
- Credit Details: AMA PRA Category 1 Credit™️: 1.0 hours
Nursing: 1.0 hours
Pharmacy: 1.0 hours - MOC Credit Details: ABS - 1.0 Point; Credit Type(s): Accredited CME (ABS)
ABPATH - 1.0 Point; Credit Type(s): Lifelong Learning (ABPATH)
ABIM - 1.0 Point; Credit Type(s): Medical Knowledge (ABIM)
ABS - 1.0 Point; Credit Type(s): Self-Assessment (ABS)
ABP - 1.0 Point; Credit Type(s): Lifelong Learning and Self-Assessment (ABP) - Commercial Support: No
- Activity Type: Enduring Material
- CME Finder Type: Online Learning
- Fee to Participate: Variable
- Measured Outcome: Learner Knowledge, Learner/Team Competence
- Provider Ship: Directly Provided
- Registration: Open to all
- Specialty: Adolescent Medicine, Gastroenterology, General Pediatrics, General Surgery, GI (incl. Liver, Pancreas, Bilary), Internal Medicine, Neonatal-Perinatal Medicine, Pediatric Gastroenterology
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